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MIF-2 in distinction to MIF seems to lack the mandatory CXCR-interacting motifs needed for activation, and it is believed to exert a more selective action in activating the tissue-protective CD74 signaling pathway. That said, MIF triggers the CD74/CD44/AMPK receptor signaling pathway, which promotes glucose uptake in cardiomyocytes and protects the guts in the course of ischemia-reperfusion damage (ninety three, ninety four). Further experiments are demanded to determine the probable of MIF/MIF-two to be a treatment strategy to safeguard the center in opposition to ischemic injuries.
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based mostly on these conclusions, improving the CD74 pathway may possibly signify a singular therapy strategy for selling healing in IBD. Though, obtaining the correct ligand to encourage CD74 may well current a challenge.
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destructive regulation of intrinsic apoptotic signaling pathway in response to DNA injury by p53 class mediator
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The pharmacological and genetic inhibition of MIF, or maybe the blockade of CD74–MIF interactions, reduces tumorigenesis in multiple genitourinary cancers by means of several different mechanisms.
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